Repost 2001: TBE/ALS [Clinical pathogenic peculiarities of chronic Russian tick-born encephalitis] 
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 Repost 2001: TBE/ALS [Clinical pathogenic peculiarities of chronic Russian tick-born encephalitis]

1: Zh Nevrol Psikhiatr Im S S Korsakova 2001;101(4):10-5 Related Articles,
Books, LinkOut  

[Clinical pathogenic peculiarities of chronic Russian tick-born encephalitis]

[Article in Russian]

Nadezhdina MV.

Clinical, serologic, epidemiological and neurological studies were carried out
in 23 patients (18 men, 5 women) aged 15-69 years who had chronic Russian
tick-borne encephalitis (RTE). With permanent stable hum{*filter*}immunity the fact
that these forms might develop was confirmed in 73.9% of the cases. In 26.1% of
the patients were found to have incomplete vaccination and vaccine therapy
combined with RTE viral infection. The following conditions underlay
chronization were observed in all forms of acute RTE: fever in 4.3%, meningitis
in 30.4%, focal lesion in 43.5% (the encephalitic syndrome in 8.7%,
poliomyelitis in 8.7%, encephalopoliomyelitis in 26.1%). Initially progressive
course was found in 21.7% of the cases. There was a primary rise of the
hyperkinetic syndrome after the meningeal and focal forms of RTE with the
encephalitic syndrome; while development of the amyotrophic syndrome (ATS) was
found after the focal form with the poliomyelitic and encephalopoliomyelitic
syndrome. The amyotrophic lateral sclerosis (ALS) syndrome occurs as both
primary progressive RTE and ATS transformation independently of the initial RTE
form. Latent periods of development of ATS and ALS syndrome were virtually
equal (mean 1.4 years) and did not depend on the initial form of acute RTE. The
duration of development of latent chronic forms makes it necessary to follow up
RTE patients for at least 3 years.

PMID: 11490426 [PubMed - indexed for MEDLINE]

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Sun, 05 Sep 2004 12:53:05 GMT
 Repost 2001: TBE/ALS [Clinical pathogenic peculiarities of chronic Russian tick-born encephalitis]


Fri, 19 Jun 1992 00:00:00 GMT
 Repost 2001: TBE/ALS [Clinical pathogenic peculiarities of chronic Russian tick-born encephalitis]
Okay, I am confused. (So what else is new?) I didn't know ticks could carry
viruses.

<<Clinical, serologic, epidemiological and neurological studies were carried
out
in 23 patients (18 men, 5 women) aged 15-69 years who had chronic Russian
tick-borne encephalitis (RTE). With permanent stable hum{*filter*}immunity the fact
that these forms might develop was confirmed in 73.9% of the cases. In 26.1% of
the patients were found to have incomplete vaccination and vaccine therapy
combined with RTE viral infection.>>

What sort of vaccine do they have against this virus? Anyone know?
Thanks,
 Ann - OH



Mon, 06 Sep 2004 00:15:16 GMT
 Repost 2001: TBE/ALS [Clinical pathogenic peculiarities of chronic Russian tick-born encephalitis]
Yes, Ann, that is what I have been hammering away at with many of my
"off-topic" posts lately.  Not only do ticks carry the TBE virus but they also
carry the Powassan Encephalitis virus, the SLE and the newly discovered (and
pathetically named) DTV, "Deer Tick Virus".  They are all flaviviruses, like
the most well-known West NIle Virus, like dengue and like TBE.  An latent
period after a brief flulike initial infection happens in some viral
encepahlitis with a more progressive form as detailed above.  Motor neuron
damage.  MND.  

The Russians haev been studying coinfection with Lyme and TBE and have found
that treatment with antibiotics often accelerates the progression of TBE/ALS
which matches what happens with our ALS/Lyme people.  But the Russians have
found some antibiotics do not do this- they are studying this i n detail.  Also
Ledum in vitro was found to inactivate the TBE.

ANyway, who knows how many of us have a flavivirus along with LYme.  It can be
latent for years and is a very slow virus.  

Flaviviruses can also be transmitted through unpasteurized infected MILK.

!!!



Mon, 06 Sep 2004 00:39:36 GMT
 
 [ 4 post ] 

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