Amalgams are the principle
source of exposure in the population.

Any chemist can put amalgams in solution and measure mercury vapor
evaporating from the solution.  If the solution is heated or acidic or
if you place another metal in the solution then more vapor will be
released.

Dentists are required by law to dispose of s{*filter*}amalgam as a
hazardous waste. So by federal law even the mercury alloy is too
hazardous to flush down the toilet.

Either the federal government is overreacting in response to s{*filter*}
amalgam disposal or it is underreacting in response to dentists
placing that amalgam in the mouth.

Mercury amalgam cannot be both hazardous and safe.  One of the
regulatory policies must be in error: either the 19th-century dental
practice is wrongheaded, or the late-20th-century amalgam disposal
practice is wrongheaded.

It is interesting to note that no pro-amalgamists have pointed out the
hysteria in the government's policy on s{*filter*}amalgam disposal.

So the underlying sentiment in the pro-amalgamist seems to be a deep
trust in old traditions and the intelligence of bureacracies, over and
against a consistency of thought and logic demanded by individuals.
When urinary excretions and {*filter*} measurements prove themselves to be
erorenous indicators of mercury body burden, this doesn't cause the
pro-amalgamist to question the Science supporting amalgam safety.  He
cannot believe that a poison such as mercury can really hazardous when
placed in the mouth.  So now he must look at the particular compounds
in which mercury may find itself in the body, with the effort of
minimizing its toxicity, to maintain his believe in the wisdom of
19th-century dentists and the bureacratic mind.

Source:             University Of Kentucky Medical Center
Date Posted:    Wednesday, February 10, 1999
Web Address:   http://www.sciencedaily.com/releases/1999/02/990210065946.htm

----------------------------------------------------------------------------
----

Mercury In Dental Fillings Does Not Appear To Cause Alzheimer's, According
To University Of Kentucky Study
LEXINGTON, KY (Feb. 8, 1999) - Mercury used in dental fillings does not
appear to cause Alzheimer's disease, according to a new study by University
of Kentucky researchers. Results of the study are published in the lead
article in today's Journal of the American Dental Association.
The study compared mercury levels in autopsied brains, and dental amalgam
status and history in Alzheimer's disease subjects as well as control
subjects. The researchers found no significant association of Alzheimer's
disease with the number, surface area or history of dental amalgams.

"Our key finding is that there is no relationship whatsoever between mercury
found in the brain and amalgam," said Stanley Saxe, D.M.D., one of the study
's authors and a professor emeritus of periodontics and geriatric dentistry
in the UK College of Dentistry. "Although very small amounts of mercury are
released from dental amalgam - generally when rubbed or abraded due to
brushing or eating - it is not taken up by the brain."

Funded by a grant from the National Institutes of Health, the study was
prompted by scientific controversy on the topic that has been brewing for
several years.

The study, which began in 1991, was a collaborative effort among researchers
from the UK Sanders-Brown Center on Aging and the UK College of Dentistry.
Saxe and William Markesbery, M.D., director of the Sanders-Brown Center on
Aging, led the research team. "The uniqueness of the collaborative effort
between the departments of chemistry, statistics, the College of Dentistry
and leadership from the Center on Aging, made this research possible and
successful," Markesbery said.

"This is the only study that has looked at this question in a large group of
people," Saxe said.

Dental amalgam has been used since the early 1830s and is considered an
excellent restorative material in dentistry because of its strength and
durability.

However, because dental amalgam is comprised of 50 percent mercury, a
neurotoxin, it has been the subject of continuing controversy as a possible
public health risk.

"The fact that there was no differential found in brain mercury levels due
to dental amalgams is very exciting news for the dentistry profession," Saxe
said.

http://www.sciencedaily.com/releases/1999/02/990210065946.htm

 The University of Kentucky is in DEANIAL, DENILE, DENIALE!!

carabelli

Hardy har har.

I demand to know what time it was when he posted this! Was it in the middle of
the night and he suddenly got up?????...:-)

A BIG OOOPISE HAS OCCURED!

http://www.***.com/ #dental amalgam

Vaughn left OUT this part,,,,,,,,,,,,,,,,,,

Imagine that,,,,,,,,and just last week,  he posted.

The Journal of Occupational and Environmental Medicine is indeed a serious
publication that I have been following intermittantly for the last couple of
years.  I agree with Jan that everyday occupational exposure to small amounts
of toxins can add up to a serious problem over many years of exposure.  I have
little doubt that dentists should also be concerned aboutoccupational respitory
problems.  Thankfully, today's dentists are exposed to much less mercury than
in the past.  I will be very interested in reading that article when it is
available. While looking on the journal's website, I found the below February
article on mercury exposure.  The article discusses the relationship between
fish consumption and mercury levels and discusses the validity of
"alternative"methods of mercury testing.  I wonder why Jan did not bother to
bring this one to our attention?

Vaughn

Of course the February article was not what was being discussed.

Moving on.

I wonder why Vaughn didn't bring this to our attention?,,,,,,,,,,,,,,,,*;*

******Contrast this study published in the Journal of the American Dental
Association with previous studies by the research scientists in this group when
dentists were not involved******

Brain trace elements in Alzheimer's disease.
Neurotoxicology 1986 Spring;7(1):195-206
Ehmann WD, Markesbery WR, Alauddin M, Hossain TI, Brubaker EH
    Instrumental neutron activation analysis has been used to determine the
concentrations of 16 elements in selected brain regions and separated gray- and
white-matter specimens from histologically verified Alzheimer's disease (AD)
and age-matched control patients. Significantly different (p less than 0.05)
mean concentrations of Br, Cl, Cs, Hg, N, Na, P, and Rb were observed in AD
bulk brain samples compared to controls, while no significant differences were
observed for Ag, Co, Cr, Fe, K, Sb, Sc, and Se. The differences that are most
persistent and largest in magnitude for the pooled bulk samples, males and
females, left and right hemispheres, and separated gray and white matter are
the elevation of Br and Hg and the depletion of Rb in AD compared to controls.
Significant interelement correlations for the latter elements in both AD and
control brains are also documented. Based on these studies, the possibility of
an etiological role for trace elements in AD clearly deserves further
investigation.  
[]
Regional brain trace-element studies in Alzheimer's disease.
Neurotoxicology 1988 Spring;9(1):1-7
Thompson CM, Markesbery WR, Ehmann WD, Mao YX, Vance DE
Department of Chemistry, University of Kentucky, Lexington 40506.
    Alzheimer's disease (AD) brain trace-element imbalances in the amygdala,
hippocampus and nucleus basalis of Meynert (nbM) are found in most cases to be
consistent with those previously reported in samples derived principally from
AD cerebral cortex (Ehmann et al., 1986). The elevation of mercury in AD nbM,
as compared to age-matched controls, is the largest trace-element imbalance
observed to date in AD brain. In addition to the general confirmation of
imbalances for Cs, Hg, N, Na, P, and Rb noted previously in cerebral cortex
samples, imbalances for Fe, K, Sc, and Zn were observed in two regions and one
region also exhibited imbalances for both Co and Se. Persistent imbalances for
the univalent cations Na, K, Rb and Cs support arguments for a membrane
abnormality in AD. The data presented here also provide the first comprehensive
simultaneous multi-element determinations in both control and AD nbM.
[]
Trace element imbalances in hair and nails of Alzheimer's disease patients.
Neurotoxicology 1988 Summer;9(2):197-208
Vance DE, Ehmann WD, Markesbery WR
Department of Chemistry, University of Kentucky, Lexington 40506.
    The concentrations of 17 elements in the hair and nails of 180 Alzheimer's
disease (AD) and control subjects have been determined by instrumental neutron
activation analysis (INAA). Comparisons of trace element levels of properly
matched AD and control groups revealed significant imbalances in the
concentrations of six elements (Br, Ca, Co, Hg, K, and Zn) between disease and
control groups. It is noteworthy that each of these has previously been shown
by our group, or others, to be altered in some AD brain region(s). Geometric
means for each element in both hair and nails of AD and control subjects are
presented, and significant differences noted. The significance of these
alterations with regard to the possible role of trace elements in the etiology
of AD is discussed.
[]
Trace element imbalances in isolated subcellular fractions of Alzheimer's
disease brains.
Brain Res 1990 Nov 12;533(1):125-31
Wenstrup D, Ehmann WD, Markesbery WR
Department of Chemistry, University of Kentucky, Lexington.
    Concentrations of 13 trace elements (Ag, Br, Co, Cr, Cs, Fe, Hg, K, Na, Rb,
Sc, Se, Zn) in isolated subcellular fractions (whole brain, nuclei,
mitochondria, microsomes) of temp{*filter*}lobe from autopsied Alzheimer's disease
(AD) patients and normal controls were determined utilizing instrumental
neutron activation analysis. Comparison of AD and controls revealed elevated Br
(whole brain) and Hg (microsomes) and diminished Rb (whole brain, nuclear and
microsomes), Se (microsomes) and Zn (nuclear) in AD. The elevated Br and Hg and
diminished Rb are consistent with our previous studies in AD bulk brain
specimens. Comparison of element ratios revealed increased Hg/Se, Hg/Zn and
Zn/Se mass ratios in AD. Se and Zn play a protective role against Hg toxicity
and our data suggest that they are utilized to detoxify Hg in the AD brain.
Overall our studies suggest that Hg could be an important toxic element in AD.
Whether Hg deposition in AD is a primary or secondary eventremains to be
determined.
[]
Imbalances of trace elements related to oxidative damage in Alzheimer's disease
brain.
Neurotoxicology 1998 Jun;19(3):339-45
Cornett CR, Markesbery WR, Ehmann WD
Department of Chemistry, University of Kentucky, Lexington 40506-0055, USA.
    Four elements that have been implicated in free-radical-induced oxidative
stress in Alzheimer's disease (AD) were measured by instrumental neutron
activation analysis (INAA) in seven brain regions from 58 AD patients and 21
control subjects. A statistically significant elevation of iron and zinc was
observed in multiple regions of AD brain, compared with controls. Mercury was
elevated in AD in most regions studied, but the high variability of mercury
levels in both AD and control subjects prevented the AD-control difference from
reaching significance. Selenium, a protective agent against mercury toxicity,
was significantly elevated only in AD amygdala. The elevation of iron and zinc
in AD brain has the potential of augmenting neuron degeneration through free
radical processes.
[]
More studies  published by other research scientists (with no dentist's
involvement) showing that mercury is elevated in AD
[]
Increased {*filter*} mercury levels in patients with Alzheimer's disease.
J Neural Transm 1998;105(1):59-68
Hock C, Drasch G, Golombowski S, Muller-Spahn F, Willershausen-Zonnchen B,
Schwarz P, Hock U, Growdon JH, Nitsch RM
Department of Psychiatry, University of Basel, Switzerland.
    Alzheimer's disease (AD) is a common neurodegenerative disorder that leads
to dementia and death. In addition to several genetic parameters, various
environmental factors may influence the risk of getting AD. In order to test
whether {*filter*} levels of the heavy metal mercury are increased in AD, we
measured {*filter*} mercury concentrations in AD patients (n = 33), and compared
them to age-matched control patients with major depression (MD) (n = 45), as
well as to an additional control group of patients with various non-psychiatric
disorders (n = 65). {*filter*} mercury levels were more than two-fold higher in AD
patients as compared to both control groups (p = 0.0005, and p = 0.0000,
respectively). In early onset AD patients (n = 13), {*filter*} mercury levels were
almost three-fold higher as compared to controls (p = 0.0002, and p = 0.0000,
respectively). These increases were unrelated to the patients' dental status.
Linear regression analysis of {*filter*} mercury concentrations and CSF levels of
amyloid beta-peptide (A beta) revealed a significant correlation of these
measures in AD patients (n = 15, r = 0.7440, p = 0.0015, Pearson type of
correlation). These results demonstrate elevated {*filter*} levels of mercury in AD,
and they suggest that this increase of mercury levels is associated with high
CSF levels of A beta, whereas tau levels were unrelated. Possible explanations
of increased {*filter*} mercury levels in AD include yet unidentified environmental
sources or release from brain tissue with the ...

read more »

posted this! Was it in the middle of

after hosting the after prom party last Friday, I found that I can have fun
ANITIME!!!

carabelli - who intends to post all misspellings in caps from now on -
unless I miss it.

Uh huh,,,,,,,,,

LOL.

******Contrast this study published in the Journal of the American Dental
Association with previous studies by the research scientists in this group when
dentists were not involved******

Brain trace elements in Alzheimer's disease.
Neurotoxicology 1986 Spring;7(1):195-206
Ehmann WD, Markesbery WR, Alauddin M, Hossain TI, Brubaker EH
    Instrumental neutron activation analysis has been used to determine the
concentrations of 16 elements in selected brain regions and separated gray- and
white-matter specimens from histologically verified Alzheimer's disease (AD)
and age-matched control patients. Significantly different (p less than 0.05)
mean concentrations of Br, Cl, Cs, Hg, N, Na, P, and Rb were observed in AD
bulk brain samples compared to controls, while no significant differences were
observed for Ag, Co, Cr, Fe, K, Sb, Sc, and Se. The differences that are most
persistent and largest in magnitude for the pooled bulk samples, males and
females, left and right hemispheres, and separated gray and white matter are
the elevation of Br and Hg and the depletion of Rb in AD compared to controls.
Significant interelement correlations for the latter elements in both AD and
control brains are also documented. Based on these studies, the possibility of
an etiological role for trace elements in AD clearly deserves further
investigation.  
[]
Regional brain trace-element studies in Alzheimer's disease.
Neurotoxicology 1988 Spring;9(1):1-7
Thompson CM, Markesbery WR, Ehmann WD, Mao YX, Vance DE
Department of Chemistry, University of Kentucky, Lexington 40506.
    Alzheimer's disease (AD) brain trace-element imbalances in the amygdala,
hippocampus and nucleus basalis of Meynert (nbM) are found in most cases to be
consistent with those previously reported in samples derived principally from
AD cerebral cortex (Ehmann et al., 1986). The elevation of mercury in AD nbM,
as compared to age-matched controls, is the largest trace-element imbalance
observed to date in AD brain. In addition to the general confirmation of
imbalances for Cs, Hg, N, Na, P, and Rb noted previously in cerebral cortex
samples, imbalances for Fe, K, Sc, and Zn were observed in two regions and one
region also exhibited imbalances for both Co and Se. Persistent imbalances for
the univalent cations Na, K, Rb and Cs support arguments for a membrane
abnormality in AD. The data presented here also provide the first comprehensive
simultaneous multi-element determinations in both control and AD nbM.
[]
Trace element imbalances in hair and nails of Alzheimer's disease patients.
Neurotoxicology 1988 Summer;9(2):197-208
Vance DE, Ehmann WD, Markesbery WR
Department of Chemistry, University of Kentucky, Lexington 40506.
    The concentrations of 17 elements in the hair and nails of 180 Alzheimer's
disease (AD) and control subjects have been determined by instrumental neutron
activation analysis (INAA). Comparisons of trace element levels of properly
matched AD and control groups revealed significant imbalances in the
concentrations of six elements (Br, Ca, Co, Hg, K, and Zn) between disease and
control groups. It is noteworthy that each of these has previously been shown
by our group, or others, to be altered in some AD brain region(s). Geometric
means for each element in both hair and nails of AD and control subjects are
presented, and significant differences noted. The significance of these
alterations with regard to the possible role of trace elements in the etiology
of AD is discussed.
[]
Trace element imbalances in isolated subcellular fractions of Alzheimer's
disease brains.
Brain Res 1990 Nov 12;533(1):125-31
Wenstrup D, Ehmann WD, Markesbery WR
Department of Chemistry, University of Kentucky, Lexington.
    Concentrations of 13 trace elements (Ag, Br, Co, Cr, Cs, Fe, Hg, K, Na, Rb,
Sc, Se, Zn) in isolated subcellular fractions (whole brain, nuclei,
mitochondria, microsomes) of temp{*filter*}lobe from autopsied Alzheimer's disease
(AD) patients and normal controls were determined utilizing instrumental
neutron activation analysis. Comparison of AD and controls revealed elevated Br
(whole brain) and Hg (microsomes) and diminished Rb (whole brain, nuclear and
microsomes), Se (microsomes) and Zn (nuclear) in AD. The elevated Br and Hg and
diminished Rb are consistent with our previous studies in AD bulk brain
specimens. Comparison of element ratios revealed increased Hg/Se, Hg/Zn and
Zn/Se mass ratios in AD. Se and Zn play a protective role against Hg toxicity
and our data suggest that they are utilized to detoxify Hg in the AD brain.
Overall our studies suggest that Hg could be an important toxic element in AD.
Whether Hg deposition in AD is a primary or secondary eventremains to be
determined.
[]
Imbalances of trace elements related to oxidative damage in Alzheimer's disease
brain.
Neurotoxicology 1998 Jun;19(3):339-45
Cornett CR, Markesbery WR, Ehmann WD
Department of Chemistry, University of Kentucky, Lexington 40506-0055, USA.
    Four elements that have been implicated in free-radical-induced oxidative
stress in Alzheimer's disease (AD) were measured by instrumental neutron
activation analysis (INAA) in seven brain regions from 58 AD patients and 21
control subjects. A statistically significant elevation of iron and zinc was
observed in multiple regions of AD brain, compared with controls. Mercury was
elevated in AD in most regions studied, but the high variability of mercury
levels in both AD and control subjects prevented the AD-control difference from
reaching significance. Selenium, a protective agent against mercury toxicity,
was significantly elevated only in AD amygdala. The elevation of iron and zinc
in AD brain has the potential of augmenting neuron degeneration through free
radical processes.
[]
More studies  published by other research scientists (with no dentist's
involvement) showing that mercury is elevated in AD
[]
Increased {*filter*} mercury levels in patients with Alzheimer's disease.
J Neural Transm 1998;105(1):59-68
Hock C, Drasch G, Golombowski S, Muller-Spahn F, Willershausen-Zonnchen B,
Schwarz P, Hock U, Growdon JH, Nitsch RM
Department of Psychiatry, University of Basel, Switzerland.
    Alzheimer's disease (AD) is a common neurodegenerative disorder that leads
to dementia and death. In addition to several genetic parameters, various
environmental factors may influence the risk of getting AD. In order to test
whether {*filter*} levels of the heavy metal mercury are increased in AD, we
measured {*filter*} mercury concentrations in AD patients (n = 33), and compared
them to age-matched control patients with major depression (MD) (n = 45), as
well as to an additional control group of patients with various non-psychiatric
disorders (n = 65). {*filter*} mercury levels were more than two-fold higher in AD
patients as compared to both control groups (p = 0.0005, and p = 0.0000,
respectively). In early onset AD patients (n = 13), {*filter*} mercury levels were
almost three-fold higher as compared to controls (p = 0.0002, and p = 0.0000,
respectively). These increases were unrelated to the patients' dental status.
Linear regression analysis of {*filter*} mercury concentrations and CSF levels of
amyloid beta-peptide (A beta) revealed a significant correlation of these
measures in AD patients (n = 15, r = 0.7440, p = 0.0015, Pearson type of
correlation). These results demonstrate elevated {*filter*} levels of mercury in AD,
and they suggest that this increase of mercury levels is associated with high
CSF levels of A beta, whereas tau levels were unrelated. Possible explanations
of increased {*filter*} mercury levels in AD include yet unidentified environmental
sources or release from brain tissue with the advance in neuronal death.
[]
Metals and trace elements in plasma and cerebrospinal fluid in normal aging and
Alzheimer's disease.
J Neural Transm Park Dis Dement Sect 1991;3(4):231-58
Basun H, Forssell LG, Wetterberg L, Winblad B.
Department of Geriatric Medicine, Huddinge Hospital, Sweden.
    Cerebro-spinal fluid (CSF) and {*filter*} levels of aluminum, cadmium, calcium,
copper, lead, magnesium, and mercury were studied in 24 subjects with dementia
of the Alzheimer type (DAT) and in 28 healthy volunteers. Furthermore, arsenic,
bromine, chrome, iron, manganese, nickel, rubidium, selenium, strontium, and
zinc were measured only in {*filter*}. There were significant changes in the DAT
group when compared to the controls. The plasma levels of aluminum, cadmium,
mercury and selenium were increased and the contents of iron and manganese were
lower in the DAT group as compared to control subjects. In CSF there were low
levels of cadmium and calcium and increased content of copper in DAT cases.
Iron and zinc levels in {*filter*} and calcium in both {*filter*} and CSF of DAT patients
correlated with memory and cognitive functions. Iron, manganese and strontium
levels of DAT sufferers in {*filter*} and aluminum in CSF were related with changes
in behavior.
[]
    From these results one must conclude that mercury levels are elevated in
the brain and tissues of Alzheimer's disease victims ONLY when dentists are NOT
involved in the studies.
[]
Other studies by this same group of dentists
[]
Dental amalgam and cognitive function in older women: findings from the Nun
Study.
J Am Dent Assoc 1995 Nov;126(11):1495-501
Saxe SR, Snowdon DA, Wekstein MW, ...

read more »

The typo and and misspelling squad meets at 4:00 AM sharp. Bring weapons.

Jan

The time of the posting is contained in the header.

No, I copied the entire article and it contained no references to crank,
anti-amalgam websites.

...snip...

     As I have told you before, amalgam is your subject, not mine.  I posted
a relevant article that I think that you and others should consider, that
brings no obligation to post all OTHER relevant information that I might be
able to find.  That would bog down the Internet.

     No; YOU contrast the studies.  If you feel the need to refute the
information that I posted, the proper way is to use your own logic backed up
by references to reputable sources.  You can't simply list or copy the
sources and leave the thinking to others.  Argue on the basis of research
methods and scientific information.  You can't simply drown out information
that does not fit within your belief system by flooding us with "approved"
information, you have to actually tell us where the other idea is wrong.
{*filter*} theories are not very impressive; if that is the best you have to
offer, don't bother..

The rest is snipped to save bandwidth.  It is all available in the previous
post in this thread.

Vaughn

 After being diagnosed with PN Jan did research and became convinced
that she had mercury poisoning due to amalgams. She then describes a
rapid and progressive deterioration in her health and thought she was
going to DIE. Then within HOURS of having SOME of the amalgams removed
she posts on usenet that she felt better than she had in two years!!!
And she did not have the rest of her amalgams out for SEVERAL days at
the time she felt better than she had in two years!!!!

 And given the fact that she reports this rapid decline in health and
being close to death, feeling better than she had in two years would
be an incredibly dramatic improvement in health.

And the above is COMPLETELY true if we are to believe Jan Drew since
SHE is the one who provided this information and I have quoted Jan
Drew to prove that she said it. So if Jan says that I lied then that
must mean that SHE lied because I am only repeating her OWN words!!!

And yet she blatantly lies on sci.med.dentistry when she states that
the reason that she knows it was the mercury poisoning is because she
started regaining her health AFTER her mercury level began to drop.
This is a lie. And I could call it a lie because Jan has ADMITTED that
she felt better than she had in two years before she had all the
amalgams removed. And yet despite this being pointed out to Jan, she
remains in a kind of psychotic denial and could only respond to my
comments by personally attacking me. It would be funny if it were not
so sad.

Aloha,

Rich

------------------------------------------------
------------------------------------------------

The best defense to logic is ignorance.

I have an opinion about crank, junk-science websites, regardless of topic.
You don't need them Jan, you have what is between your ears and you have
PubMed.  Do it right and perhaps you might actually convince someone.

Jan, you seem to have cut out the below paragraph.  Now cutting it out is
perfectly OK, but you should have bothered to READ it first!

"YOU contrast the studies.  If you feel the need to refute the
information that I posted, the proper way is to use your own logic backed up
by references to reputable sources.  You can't simply list or copy the
sources and leave the thinking to others.  Argue on the basis of research
methods and scientific information.  You can't simply drown out information
that does not fit within your belief system by flooding us with "approved"
information, you have to actually tell us where the other idea is wrong.
{*filter*} theories are not very impressive; if that is the best you have to
offer, don't bother.."

     Again, you offer your theory of the dental {*filter*}.  You did not
bother to take the slightest look or make any comment on the science
involved.  I totally reject that crap.  If you want to do something like
that and do it right, look into something called a "meta-study".  You could
draw ALL of the studies on a given subject and within a given time period
and compare the way the subject is treated in some objective manner.  It is
very possible that you could be proven right.  That is; you MAY, based on
some objective criteria, find some statistically significant differences in
the way dentists and MD's handle the identical topic.  Until then, the
comparison of hand-picked papers that you are offering here is junk-science
drivel.

{*filter*} theory drivel clipped...

Vaughn

So you are saying that Dr. Boyd Haley is a crank, and his work is junk-science?

FYI, PubMed is written by organized medicine and dentistry.

I READ it!

WRONG! I do NOT write the studies. It is done by researchers, MD's scientists
and DDS's. I just post it.

I don't need to put it in my own words Vaughn, that's already done. The
websites DO have references. What do you consider reputable? Dr Haley at the
currect time is the top researcher and you call his work junk science. Have you
read any of the long list, he has provided?

Sure I can! I am just providing the many risks. You should be able to
understand that.

That's what I provide.

That's EXACTLY what those in denial do!

I've been doing that now for three years. You can lead a horse to water, but
you can't make him drink.

I'm more interested in truth, than impressions.

Point made!

Simply wrong. I am getting tired of saying the same things over and over.

Point made!

Un huh,,,,,,,,,,you mean ONLY an organized medicine and dentistry study.

Translation: Do it MY way,,,,,,,,,,,,

So you can say. I don't believe that CRAP.

Yes, that will happen after I'm gone, and how many before me have been met with
your very same attitude, it is CRAP. This should have been proven LONG ago!

 That is; you MAY, based on

find some statistically significant differences in

That's been done for me!

Hand picked? I have reported all that is available on the web. I have many many
websites, you call ALL of them drivel.

What I find strange, is you want to spread the news of the dangers your wife
suffers and with VERY good reason, but when I try to warn other suffering
people of the risks, THAT I SUFFERED,,,,,,,,,it is drivel.

That sucks Vaughn.

Jan

More {*filter*} crap.

     Go back over your last few posts, that is exactly what YOU have been
doing.  You have not offered a single idea of your own, you have still not
addressed the original article that I posted, you just attempted to drown it
out!

Then address the original article and give up on the {*filter*} stuff.

More {*filter*} crap.

     Because they ARE, you do not need them.  Use science and that grey
stuff between your ears.  One good logical point driven home, properly
supported by the science,  trumps listing a thousand {*filter*}websites.

     Because you insist on spreading crap.  If I shared your concerns about
amalgam to the extent that I felt I had to crusade on the Internet, I would
want maximum distance from the likes of you.  You have negative impact.

      I have told you many times nicely and not so nicely, you do not
listen.  If the "Organized dentistry" conspiricy theory is the best you can
offer, your back is truly up against the wall and you may as well give it
up; there is nothing left.

Poor Jan!

Goodnight Jan:
Vaughn

Thanks for the {*filter*}discussion and insults.

Jan