EDTA Chelation Therapy 
Author Message
 EDTA Chelation Therapy

I note the absence of recent discussion on EDTA chelation therapy, so I
will re-introduce the subject with a personal anecdote.

I have had a casual interest in the subject for several years, but became
more interested in late November when, after resuming taking tenormin
for high {*filter*} pressure, I developed angina pectoris (heart pains). I
decided that, facing bypass surgery, it might be worth giving chelation
therapy a try.

(Note: EDTA is disodium ethylene diamine tetraacetic acid, typically
administered by injection of a solution of 500ml Ringers, consisting of
10-15cc EDTA, 10-15cc ascorbic acid (Vitamin C), 2cc lidocaine, 3cc
magnesium, 2cc B-complex, administered over a period of about 3 hours.)

After 9 treatments, over a period of 2 months, I can report the following:

BEFORE                              AFTER

Angina upon walking more than       Slight discomfort if I run or rapidly
about 100 feet briskly.             climb stairs, but no longer from walking.

5, slowing rising.      BP of 132/84, falling.
Drowsiness, fatigue, shortness      Alert, well-rested, no shortness of
of breath.                          breath.

{*filter*}ly discomfort in legs.         No more discomfort.

Mild arthritis, especially in       No more arthritis symptoms.
hands.

Muscle stiffness, creaking joints.  Muscles more limber, no more creaking.

Calcium deposit on knuckle.         Calcium deposit shrinking rapidly.

Varicose veins in feet, ankles.     Varicose veins reduced by about 40%.

Cold hands, feet.                   Warm hands, feet.

There have also been other general improvements in health and comfort.

Tenormin was discontinued prior to beginning chelation therapy, and has
note be resumed. (One of the side-effects of tenormin is heart failure!).
Vasotec had other undesirable side-effects.

The chelation therapy is self-administered. It is supplemented with a
course of vitamin and mineral tablets, and I have begun to eliminate
sources of free-radical production from my diet to the extent possible
for someone who eats out all the time.

Chelation therapy has been attacked on the grounds that it does not
reduce arteriosclerosis plaque or have a lasting effect. Its defenders
claim that whatever its effect on plaque, it seems to reduce the
impact of plaque, and that its effects are indeed lasting with some
maintenance treatments at the rate of say, one every two months or so.

Based on my experience, I must side with the defenders. There is not\
placebo effect here. I know how to counter for that.

Comments would be welcome.

More information on chelation therapy can be obtained from:

American Academy of Medical Preventics
6151 W Century Blvd #1114
Los Angeles, CA 90045

International Chelation Research Foundation
3218 Pauline Dr
Chevy Chase, MD 20815

These addresses may be out of date. I haven't checked them lately.



Sat, 20 Jul 1996 11:55:25 GMT
 EDTA Chelation Therapy

Quote:

>5, slowing rising.      BP of 132/84, falling.

This line got garbaged. It should read

BP of 142/105, slowing rising.       BP of 132/84, falling.

Quote:
>Based on my experience, I must side with the defenders. There is not\

This should read

Based on my experience, I must side with the defenders. There is no



Sat, 20 Jul 1996 12:04:46 GMT
 EDTA Chelation Therapy

Quote:

>I note the absence of recent discussion on EDTA chelation therapy, so I
>will re-introduce the subject with a personal anecdote.
>I have had a casual interest in the subject for several years, but became
>more interested in late November when, after resuming taking tenormin
>for high {*filter*} pressure, I developed angina pectoris (heart pains). I
>decided that, facing bypass surgery, it might be worth giving chelation
>therapy a try.

[nature of EDTA treatment deleted]

Quote:
>After 9 treatments, over a period of 2 months, I can report the following:

>BEFORE                              AFTER

[deleted; suffice it to say that "AFTER" is better]

Quote:
>There have also been other general improvements in health and comfort.
>Tenormin was discontinued prior to beginning chelation therapy, and has
>note be resumed. (One of the side-effects of tenormin is heart failure!).
>Vasotec had other undesirable side-effects.

Hold on here.  You started taking Tenormin, developed angina, stopped
taking Tenormin, angina went away.  Why is chelation getting the
credit?

Quote:
>The chelation therapy is self-administered. It is supplemented with a
>course of vitamin and mineral tablets, and I have begun to eliminate
>sources of free-radical production from my diet to the extent possible
>for someone who eats out all the time.

Pretty far from normal EDTA administration.  Can anyone comment on how
much EDTA gets into the {*filter*}stream this way?

Quote:
>Chelation therapy has been attacked on the grounds that it does not
>reduce arteriosclerosis plaque or have a lasting effect. Its defenders
>claim that whatever its effect on plaque, it seems to reduce the
>impact of plaque, and that its effects are indeed lasting with some
>maintenance treatments at the rate of say, one every two months or so.
>Based on my experience, I must side with the defenders. There is not\
>placebo effect here. I know how to counter for that.

Countering for it in yourself is almost impossible, unless you are
carefully giving yourself unlabeled pills that are either placebos or
are not.  After all, when your biggest problem is pain or related to
pain, that's a toughie -- pain is notoriously hard to measure, and is
strongly affected by mood, etc.

Don't get me wrong -- I'm glad you're feeling better and all, but how
do we know it's the chelation and not your new diet, for example?  And
are you getting more exercise than you were before?  There are so
many variables here that attributing the results to a single cause
seems awfully shaky to me.

  -- David Wright, Hitachi Computer Products (America), Inc.  Waltham, MA

     but you're free to disagree, you poor deluded creature



Sat, 20 Jul 1996 21:00:56 GMT
 EDTA Chelation Therapy
The Life Extension Foundation's latest newsletter quotes a scientist
explaining a lot of e{*filter*}ment over the discovery that oxidation of LDL
cholesterol is the key factor that cause atheriosclerosis. This would
explain why chelation works- it buffers free radicals.


Sat, 20 Jul 1996 21:39:02 GMT
 EDTA Chelation Therapy

Quote:

>Hold on here.  You started taking Tenormin, developed angina, stopped
>taking Tenormin, angina went away.  Why is chelation getting the
>credit?

Tenormin was discontinued about 3 weeks before commencement of chelation
therapy, during which angina continued, or worsened slightly. Angina
began to get better after about the 4th chelation treatment and has been
getting better each day thereafter.

I am taking more vitamins now, but was also taking a supplement before.
This course differs mainly in including more minerals of the kind that
chelation therapy might deplete.

Eating habits haven't changed that much. Mostly less fried foods, no butter
or margarine, no salad dressing, etc. But I never ate much of those anyway.

I used to run a lot, but have not been exercising beyond short walks. I
want to get the angina down a lot more before attempting real running.

Incidentally, BP is now down to 122/82, and seems to have stablized at
that level.

Quote:
>Pretty far from normal EDTA administration.  Can anyone comment on how
>much EDTA gets into the {*filter*}stream this way?

This is normal chelation therapy practice as discussed in many books on
the subject, except that some of them add heparin, to prevent clotting.
My advisors tell me that that is unnecessary, as EDTA also prevents
clotting. I have had no problems with that.

Quote:
>are you getting more exercise than you were before?  There are so
>many variables here that attributing the results to a single cause
>seems awfully shaky to me.

True, it is not a controlled experiment. However, the results are
consistent with those obtained by others under more controlled conditions.

Just a data point.



Tue, 30 Jul 1996 09:22:06 GMT
 EDTA Chelation Therapy

Quote:
>The Life Extension Foundation's latest newsletter quotes a scientist
>explaining a lot of e{*filter*}ment over the discovery that oxidation of LDL
>cholesterol is the key factor that cause atheriosclerosis. This would
>explain why chelation works- it buffers free radicals.

Current prevailing theory among chelation therapists is that it works in
several different ways, one of the most important being that it removes
"free radical accelerators" -- mainly heavy metal ions -- which greatly
increase the rate of production of free radicals, much like an internal
source of ionizing radiation.

I have noticed that my veins, which I can examine more easily than my
arteries, since more of them are just under the surface of my skin,
seem to have gotten larger and softer since beginning chelation therapy.
If the same thing is happening to the arteries, this could explain
increased {*filter*} flow even though plaque reduction is not very great.
Might also make the plaque more permeable to the flow of oxygen.

I recall news reports of a family in Italy which is free of cardiovascular
problems, seemingly as the result of an inherited trait which involves
the production of a protein that protects against cardioascular disease.
One wonders whether that protein might operate similarly to EDTA, and
whether this might involve a gene that our ancestors had but lost, like
the gene to make Vitamin C.



Tue, 30 Jul 1996 09:31:42 GMT
 EDTA Chelation Therapy

Quote:
>I would stay away from EDTA... It's a toxin - if you drink enough of it you
>will not only remove the heavy metals floating around in your {*filter*} (if any)
>but will lower you {*filter*} calcium to dangerous levels, and generally bust up
>cell membranes (makes "holes" in membranes).  If you want to use something
>against angina or to lower the possibility of coronary thrombosis take low
>dose aspirin (if you don't have stomach ulcers).  BTW, some types of
>angina don't respond to aspirin treatment.  A plethora of scientific articles
>have shown that aspirin has some effect, though it is not the "alpha and
>omega" of cures for coronary heart disease.  New {*filter*} are continually being

Administration of EDTA is intravenous, not oral. Best evidence is that it
removes ionized but not chelated calcium, and most biologically needed
calcium is chelated. Amount removed is very small, however, and other
metals are heavily preferred to calcium. Also, I take a calcium supplement
after every treatment.

Tried aspirin. Helped a little, but not nearly enough.

You might want to take another look at EDTA chelation therapy. Note that
this is the disodium variety, not the one used in most other chelation
therapy.



Tue, 30 Jul 1996 09:39:26 GMT
 EDTA Chelation Therapy

Quote:
>    This is a method of dealing with not only heart disease,
>but high {*filter*} pressure, high cholesterol, scientifically proven
>effective by adopting changes in lifestyle alone, like reducing and/or
>managing stress as well as fat, etc.

My lifestyle already comes fairly close to such a regimen, which, while
it helps some, is not a panacea, ether. The Apollo astronauts who died
in the fire were found to have plaque buildups that, if found earlier,
would have led to them being prescribed bypass surgery. The problem with
exercise is that it enlarges the arteries without actually removing plaque,
so that if and when exercise is inevitably reduced, constriction of the
arteries that then follows can preciptate a crisis.

Work is being done on testing several candidate "drano" {*filter*}. I am
hoping chelation therapy will hold me until one of them pans out.



Tue, 30 Jul 1996 09:47:52 GMT
 
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